Why Do Purines Bond With Pyrimidines in the DNA Ladder?
There has been a lot of research done on the role of purines in the DNA structure. The purines and their derivatives are part of the DNA of all living organisms. The job of these proteins is to read the genetic code of the organism and then produce the proteins needed for further processing in the cell. Unfortunately, every time a cell divides, purines leave the bounds of the double helix and attach themselves to another protein in the process. This introduces a mutation in the genetic code, which can be read by other cells in the body, usually resulting in disease. Most people would think that mutating the DNA is terrible. Still, it provides the flexibility for an organism to develop a healthy immune system and a varied diet from its natural food.
Why do purines bond with pyrimidines in the DNA ladder? This is a question that has plagued scientists for years and one that only seems to be getting more relevant as studies continue to unravel the mysteries of human genetics. There is no doubt that purines and their molecules play an essential role in the biological processes of living things. The problem, however, is that the exact function of each purine molecule is unknown. While sequencing DNA has revolutionized the medical community, it has only scratched the surface of understanding purine function. The next few years will promise to offer greater insight into the role of these abnormal proteins in the genetic code.
One of the biggest mysteries in DNA is why purines bond with pyrimidines in the DNA ladder. One possible answer to this problem centers on a type of chemical mechanism known as “directed evolution.” This is a well-studied process in which one kind of DNA tends to become more similar to another as it is copied and passed down the family tree. This process is only applicable to deoxyribonucleic acid, or DNA, and there are only a handful of specific DNA variations that respond to this kind of “shuffling.”
Another possibility for studying why purines bond with pyrimidines in the DNA ladder concerns a process known as “genetic drift.” This happens when too many of a specific type of DNA sequence within a species occurs because there are not enough of a different kind of DNA to account for its formation. There are two different ways that this can happen. First, random mutations can occur without any chance of natural selection, which can significantly skew the genetic make-up of a species.
Genes that are passed on between closely related species can also experience genetic drift. This means that there are segments within the species that are more similar than others. Because these segments have only recently come into existence, their DNA is incredibly identical. But because new generations of organisms are constantly forming, the differences within the details can accumulate, and over time, the segments separate and form new species.
The difficulty in answering why purines bond with pyrimidines in the DNA ladder comes from the fact that researchers have yet to identify all of the genes that code for pyrimidines. Many of the genes necessary for purines to act on the pyrimidines helpfully have yet to be discovered. Much of the study has focused on understanding those genes that are most similar to pyrimidines or those that are highly conserved. Fewer genes remain to be tested for, making the relationship between the purines and pyrimidines complicated.
A third possible answer to why purines bond with pyrimidines in the DNA ladder is that purines and pyrimidines are part of a very general class of proteins called nucleic acids. These are simply chains of carbon and hydrogen atoms joined together. They are an essential part of every cell in the human body and all life on earth. The code for specific functional proteins, and sometimes, they are required to form the complete genes.
Some researchers have speculated that the specific sequence of amino acids necessary to form pyrimidines may have been introduced in a gene pool by some form of common ancestry. This idea received great scientific faith in the late nineteen sixties through the work of Dr. James M.selves. Dr. M. selves proposed that purine inheritance is passed on between species through the species of a single gene. This study provided the first evidence in support of this theory.